encore life cbd

Encore life cbd

CBD
Supports natural endocannabinoid production and activity by slowing down Anandamide’s transport into the cell, thus decreasing breakdown by FAAH.

Heart: CB1
Regulates: Inflammation, atherosclerosis

Mostly located in the periphery, especially cells associated with the immune system.

Primarily found in the brain and central nervous system, and to a lesser extent in other tissues.

CB2 Receptors

Brain: CB1
Regulates: Neurological health (pain, anxiety, mood, sleep, focus, motor skills, short-term memory)

The Endocannabinoid System plays a major role in maintaining balance in many physiologic functions. If the body suffers a lack of endocannabinoids, the result is Clinical Endocannabinoid Deficiency.

Skin: CB2
Regulates: Dermatological inflammatory conditions (acne, scar, psoriasis, eczema)

The Endocannabinoid System (ECS)

Ask us about professional CBD products you can trust.

Immune System: CB2
Regulates: The body’s natural defense system

Benefits include:

Increased consumer attention has led to emerging scientific research on the numerous benefits of hemp-derived cannabidiol.

Naturae’s Path and Dr. Brown are very proud to now offer CBD, in both tincture and gummy forms, and it is available in-office and through our online shopping – direct from manufacturer – Encore Life. If you login to Encore’s website to order products – you will need our physician code – MDB369.

Top Quality Medical CBD – Professional Line only

A Naturopathic Doctor’s approach for using CBD

Marijuana may produce mild dependence in humans. 8-12 This was shown to depend on the personality type of the addicts, 13 and can be successfully reversed by abstinence or treated by cognitive-behavioral therapy, 14 without the occurrence of major withdrawal symptoms. Cannabinoids act on brain reward processes and reward-related behaviors by a mechanism similar to that found with other addictive drugs. In animal models they enhance electrical brain-stimulation reward in the core meso-accumbens reward circuitry of the brain and neural firing of a core dopamine (DA) component and thus elevate DA circuit. In the reward-relevant meso-accumbens DA circuit. In some animal models they produce conditioned place preference (CPP) and self-administration. 15,16 Other studies, however, find THC to be a poor reinforcer, with no or little self-administration. 17

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Résumé

Parkinson’s disease (PD) is a chronic, progressive neurodegenerative disorder. The main pathological feature of PD is the degeneration of dopamine (DA)-containing neurons of the substantia nigra, which leads to severe DAergic denervation of the striatum. The irreversible loss of the DA-mediated control of striatal function leads to the typical motor symptoms observed in PD, ie, bradykinesia, tremor, and rigidity. It has been proposed that cannabinoids may have some beneficial effects in the treatment of PD. 129 In animal experiments cannabinoids provide neuroprotection against 6-hydroxydopamine toxicity in vivo and in vitro. 131

Multiple sclerosis, neuroprotection, inflammation

The abuse of other substances is influenced by the cannabinoids. The cannabinoid system is involved in alcohol-consumption behavior. Cannabinoid CB1 receptor agonists have been found to specifically stimulate alcohol intake and its motivational properties in rats. 18 The high ethanol preference of young mice is reduced by the cannabinoid receptor 1 (CB1) antagonist SR141716A (rimonabant) to levels observed in their CB1 knockout littermates. 19 Dopamine release induced by ethanol in brain was reduced by SR141716A, 20 which can explain in part the antiaddictive effect of the drug. Cocaine is another substance of abuse in whose acquisition and consolidation cannabinoids may be involved. High prevalence of alcohol dependence and cannabis dependence can be found in patients with cocaine dependences. 21 Marijuana smoking increases plasma cocaine levels and subjective reports of euphoria in male volunteers. 22,23 Furthermore, a recent genetic study found an association between an n triplet repeat polymorphism in the CB1 encoding CNR1 gene with cocaine addiction in the African-Caribbean population. 24 In another study it was found that withdrawal from repeated access or exposure to cocaine and then a reinstatement of cocaine-seeking behavior or a sensitized locomotor response to a single cocaine challenge, respectively, was potently reduced by pretreatment with rimonabant. 25 Similarly, acute administration of rimonabant blocked expression of nicotineinduced conditioned place preference. 26 Rimonabant also reduces nicotine self-administration, and may be effective not only as an aid for smoking cessation, but also in the maintenance of abstinence. 27 As the endocannabinoid system plays a role in nicotine addiction, 28 the potential of cannabinoid antagonists to treat it is self-evident. 29-31 Opiate and CB1 receptors are coexpressed in the nucleus accumbens and dorsal striatum, and the interaction between the two systems is well known. 32 The reinforcing properties of morphine and the severity of the withdrawal syndrome are strongly reduced in CB1-knockout mice 33 ; this observation opens an opportunity to treat opiate addiction with rimonabant, as noted with alcohol, cocaine, and nicotine addiction. 34,35

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